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Dr. Michail Panagiotidis serves as Professor and Department Head of the Department of Comparative Biomedical Sciences in the College of Veterinary Medicine at Mississippi State University, having joined the institution in January 2025. His academic disciplines include pharmacology, toxicology, and One Health. Previously, he was Head of the Department of Cancer Genetics, Therapeutics and Ultrastructural Pathology at the Cyprus Institute of Neurology and Genetics in Nicosia, Cyprus. Earlier roles encompass positions at Northumbria University in Newcastle upon Tyne, England, UK, and Professor and Chair of the School of Life Sciences at Heriot-Watt University in Edinburgh, Scotland, from January 2013 to August 2016.
Dr. Panagiotidis has authored numerous influential publications on oxidative stress, DNA damage, epigenetics, and carcinogenesis. Key works include 'Comet assay in human biomonitoring studies: reliability, validation, and applications' (1997, Environmental and Molecular Mutagenesis), 'Oxidative stress, DNA methylation and carcinogenesis' (2008, Cancer Letters), 'The central role of glutathione in the pathophysiology of human diseases' (2007, Archives of Physiology and Biochemistry), 'Reactive Oxygen Species (ROS)–Induced genetic and epigenetic alterations in human carcinogenesis' (2011, Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis), 'Oxidative Stress, Redox Signaling, and Autophagy: Cell Death Versus Survival' (2014, Antioxidants & Redox Signaling), and 'DNA damage induced by endogenous aldehydes: current state of knowledge' (2011, Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis). More recent contributions address taurine against oxidative stress (2021), arsenic-induced neurotoxicity (2019), and the role of isothiocyanates in cancer chemoprevention (2019). These publications demonstrate his substantial impact on understanding molecular mechanisms of disease, environmental toxicity, and potential therapeutic interventions.

Photo by Osarugue Igbinoba on Unsplash
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