Rate My Professor Alice Lee

Dr. Alice Lee is a Lecturer in the School of Pharmacy and Biomedical Sciences within the College of Health at Adelaide University. Having joined the Biosciences team in Clinical Health Sciences in 2010 as a teaching academic, she brings extensive experience in education and research. Lee obtained her PhD from the University of Adelaide, where her doctoral research investigated the importance of dietary vitamin D and calcium on bone structure and strength in both young and osteoporotic populations. Following her PhD, she held a postdoctoral position examining the effects of vitamin D supplementation in cancer-affected populations.

In teaching, Dr. Lee focuses on first-year allied health courses, including Physiology Essentials and Human Physiology, as well as nursing courses at various levels such as Human Body 2, Scientific Basis of Clinical Science, and Evidence-Based Nursing Practice. She also teaches undergraduate and postgraduate Human Neuroscience courses and has served as course coordinator for several of these. Eligible to co-supervise Masters and PhD students, she supports postgraduate research.

Dr. Lee's research centers on nutritional factors influencing bone health, including vitamin D, calcium, resveratrol, and genistein, particularly in relation to chemotherapy-induced bone loss and skeletal development. Her key publications include Su, Y. W., et al. (2023). Methotrexate Chemotherapy Causes Growth Impairments, Vitamin D Deficiency, Bone Loss, and Altered Intestinal Metabolism—Effects of Calcitriol Supplementation. Cancers, 15(17); Lee, A., et al. (2019). Individual or combination treatments with lapatinib and paclitaxel cause potential bone loss and bone marrow adiposity in rats. Journal of Cellular Biochemistry, 120(3), 4180-4191; Lee, A., et al. (2017). Effects of resveratrol supplementation on methotrexate chemotherapy-induced bone loss. Nutrients, 9(3), 255; King, T., et al. (2015). Potential effects of phytoestrogen genistein in modulating acute methotrexate chemotherapy-induced osteoclastogenesis and bone damage in rats. International Journal of Molecular Sciences, 16(8), 18293-18311; Lee, A., et al. (2014). Adequate dietary vitamin D and calcium are both required to reduce bone turnover and increased bone mineral volume. The Journal of Steroid Biochemistry and Molecular Biology, 144(Part A), 159-162. Other works cover effects of maternal hypoxia on offspring bone and parental genome impacts on fetal bone phenotype.