Rate My Professor Emma Gray

Prominent among her publications is 'Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis' (Nature Genetics, 2011; cited 3,268 times), which delineates genetic risks emphasizing cell-mediated immunity in multiple sclerosis. She co-authored 'The zebrafish reference genome sequence and its relationship to the human genome' (Nature, 2013; cited 6,576 times), establishing a key reference for comparative genomics between zebrafish and humans. Another significant paper is 'Bi-allelic Loss-of-Function CACNA1B Mutations in Progressive Epilepsy-Dystonia' (American Journal of Human Genetics, 2019; cited 76 times), reporting CACNA1B mutations linked to this disorder. Additional works include 'Pooled Sequencing of 531 Genes in Inflammatory Bowel Disease Reveals Protein-Protein Interactions' (PLoS Genetics, 2015), uncovering interactions in IBD; 'Identification of 15 new psoriasis susceptibility loci highlights the role of shared risk loci with other autoimmune diseases' (Nature Genetics, 2010); 'Genome-wide association analysis identifies three new risk loci for childhood acute lymphoblastic leukemia' (Human Molecular Genetics, 2015); 'Evidence for three genetic loci involved in both anorexia nervosa and type 1 diabetes'; 'The correlation between reading and mathematics ability at age twelve has a substantial genetic component' (Nature Communications, 2014); 'A genome-wide association analysis of a broad psychosis phenotype'; and 'AusTraits, a curated plant trait database for the Australian flora' (Nature Communications, 2021). These publications reflect her extensive involvement in collaborative genomic research projects, often in partnership with the Wellcome Trust Sanger Institute.