Rate My Professor Kay Potapov

Kay Potapov is an Assistant Research Fellow in the Department of Physiology, School of Biomedical Sciences, at the University of Otago. She is part of the Faculty of Biomedical Sciences within the Health Sciences Division. Potapov is a member of the Campbell Lab and contributes to the Heart Otago research group. Her research focuses on investigating the central neuroendocrine abnormalities of polycystic ovarian syndrome (PCOS), the leading cause of infertility in women. She holds a Bachelor of Science degree from Nagoya University. Potapov has co-authored multiple peer-reviewed journal articles and conference contributions in the fields of neuroendocrinology and neuroscience. Her work includes studies on hyperandrogenic mouse models of PCOS, examining the roles of androgen receptors in forebrain neurons and GABA neurons, progesterone receptor expression, and GnRH neuron circuits.

Key publications include: Lott, E. E. A., Prescott, M., Potapov, K., Handelsman, D. J., Glendining, K. A., & Campbell, R. E. (2025). Forebrain AR deletion restores PR expression but not reproduction in prenatally androgenised female mice. Endocrinology; Sucquart, I. E., Coyle, C., Rodriguez Paris, V., Prescott, M., Glendining, K. A., Potapov, K., et al. (2024). Investigating GABA neuron-specific androgen receptor knockout in two hyperandrogenic models of PCOS. Endocrinology, 165, bqae060; Glendining, K. A., Prescott, M., Potapov, K., Kip, E., & Campbell, R. E. (2026). Arcuate nucleus-specific progesterone receptor knockdown in female mice is sufficient to recapitulate PCOS-like phenotypes. Journal of Neuroendocrinology; Jimenez-Martin, J., Potapov, D., Potapov, K., Knopfel, T., & Empson, R. (2021). Cholinergic modulation of sensory processing in awake mouse cortex. Scientific Reports, 11, 17525; Power, E. M., et al. (2017). Motor and Cerebellar Architectural Abnormalities during the Early Disease Stage of Spinocerebellar Ataxia Type 6. Cerebellum. Conference presentations feature Lott, E., et al. (2024). Androgen receptor expression in neurons is not required to program reproductive dysfunction in polycystic ovary syndrome-like mice, at the 18th New Zealand Medical Sciences Congress; Hames, M., Potapov, K., Ohline, S. M., & Jones, P. P. (2024). Investigating the effect of CK2 phosphorylation of RyR2 on neuronal excitability, Australasian Winter Conference on Brain Research; and Deeney, E., Ibrahim, M., Potapov, K., Hughes, S., & Empson, R. (2022). Voluntary exercise restores motor performance in a mouse model of spinocerebellar ataxia type 1 (SCA1), Australasian Winter Conference on Brain Research.