Rate My Professor Megan Inglis

Megan Inglis serves as a Research Technician in the Department of Anatomy at the University of Otago in Dunedin, New Zealand. She works in the Anderson Laboratory, part of the Centre for Neuroendocrinology, which investigates the neural mechanisms regulating fertility. The lab examines how metabolic hormones such as leptin and insulin convey information about nutritional status to the brain and explores the role of RFRP neurons in modulating fertility and stress responses, including anxiety. Research in the laboratory includes chemogenetic studies on RFRP neurons and their impact on luteinizing hormone pulses and fertility in female mice. All experiments are approved by the University of Otago Animal Ethics Committee. Megan Inglis provides critical technical support, including assistance with protocols and animal facility work, as acknowledged in multiple student theses and research outputs.

Inglis has co-authored peer-reviewed publications in neuroendocrinology. These include 'Deletion of suppressor of cytokine signaling 3 from forebrain astrocytes in mice' in the Journal of Neuroscience (2016), with co-authors H. McEwan, Janette Quennell, David Grattan, and Greg M. Anderson. She is also credited for technical assistance in 'Chemogenetic Activation of RFRP Neurons Reduces LH Pulses and Fertility in Female Mice' (2024), by I.L. Sawyer and colleagues. Earlier in her career, associated with the Department of Microbiology and Immunology at the University of Otago, she contributed to research on oral probiotics and bacteriocins. Key publications from this period are 'Persistence of the Oral Probiotic Streptococcus salivarius M18 Is Dose Dependent and Megaplasmid Transfer Can Augment Their Bacteriocin Production and Adhesion Characteristics' in PLoS ONE (2013), by J.P. Burton and colleagues; 'Ubericin A, a Class IIa Bacteriocin Produced by Streptococcus uberis' in Applied and Environmental Microbiology (2007), by N.C.K. Heng and colleagues; and 'The effect of ingestion of milk supplemented with salivaricin A-producing Streptococcus salivarius on the bacteriocin-like inhibitory activity of streptococcal populations on the tongue' in FEMS Microbiology Ecology (2007), by K.P. Dierksen and colleagues. Her contributions span microbiology and neuroendocrinology, supporting research at the University of Otago.